Salt, Starch, and Hypothyroidism

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The Last Supper made in salt in Wieliczka Salt Mine (Poland). Photo taken by Adam Kumiszcza, Wieliczka-daVinciCC BY 3.0

Under the influence of thyroid hormone, the cell retains a greater proportion of magnesium and sodium, and it preferentially excretes potassium and calcium.  In Mind and Tissue, Raymond Peat, PhD talks about how the orienting reflex involves itself in satiety, where the organism pursues and integrates a food into his structure and thereby facilitates a certain period of growth that’s needed based on the current context.
Hypothyroidism promotes prolactin secretion, and subsequent hypo-osmolar conditions in the cell triggers starch cravings (a food devoid of water content) and salt (both to replace salt inadequately retained due to insufficient carbon dioxide production, and also to shift the cell toward a hyper-osmolar state.)

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6 responses to “Salt, Starch, and Hypothyroidism”

  1. tim Avatar

    So, would a person with very good thyroid status crave less salt(less sodium) and more fructose/sucrose ? A guy on the forum was telling that potassium makes the cell more sensitive to thyroid hormone, so i guess higher thyroid uptake by the cell will excrete some potassium out as a part of negative feedback loop

    1. Avatar

      With good thyroid function, the cell retains sodium and magnesium so as to diminish the need for salty or magnesium-rich foods. Potassium acts like insulin and improves glucose metabolism, which can accelerate the effects of thyroid hormone in a positive way, as well as help to lower adrenaline (during thyroid sensitization for example). All alkaline minerals (sodium, calcium, potassium and magnesium) can partially substitute for one another, and an adequate intake of all these can be protective. High potassium becomes increasingly necessary with a correlative increase in sugar consumption.

      1. tim Avatar

        “Hypothyroidism promotes prolactin secretion” — Is it possible to have hypothyroidism (TSH >2.5) and yet low prolactin ?

        1. Avatar

          Yes, but it’s unlikely. One marker of hypothyroidism can be suppressed (for example prolactin) while others can remain elevated. Estrogen, for example can be concentrated in the tissues but appear low on blood tests.

      2. tim Avatar

        “Potassium acts like insulin and improves glucose metabolism” — I didn’t find the reference to this claim in Dr Peat’s site. On googling, i found out that in standard ER practice,docs give insulin for hyperkalemia. Insulin apart from promoting glucose uptake in the cells also promote potassium uptake in the cells. I didn’t find a study where potassium loading decreased blood glucose in humans. It’s the other way around where exogenous glucose by stimulating endogenous secretion of insulin, enhances extrarenal disposal of a potassium load .

        1. Avatar

          Although epidemiological, this study has some useful info.

          “Potassium, both serum levels and to a lesser extent dietary intake levels, has been associated with incident diabetes. Lower levels of potassium have been found to be associated with a higher risk of diabetes in some studies.”

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